Overview
Anterior cruciate ligament (ACL) tear is one of the most common knee injuries, particularly in young athletes involved in pivoting sports (soccer, basketball, skiing). ACL injuries account for ~50% of all knee ligament injuries, with an incidence of ~68 per 100,000 annually, more common in females due to anatomical and biomechanical risk factors. ACL tears cause instability, impaired performance, and predispose to early osteoarthritis if untreated.
Definition
ACL: Intra-articular, extrasynovial ligament preventing anterior tibial translation and providing rotational stability.
Pivot shift: Sudden anterior subluxation of tibia during rotation; clinical sign of ACL deficiency.
Hemarthrosis: Acute intra-articular bleeding causing rapid knee swelling after ligament tear.
Non-contact injury: Injury mechanism where no direct trauma occurs, often from cutting/pivoting movements.
Anatomy & Physiology
- ACL origin: Posterior aspect of lateral femoral condyle.
- Insertion: Anterior intercondylar area of tibial plateau.
- Function: Restricts anterior translation of tibia relative to femur; stabilises knee during rotation and cutting movements.
- Bundles: Anteromedial (tight in flexion) and posterolateral (tight in extension).
ACL is the primary restraint to anterior tibial displacement.
Aetiology and Risk Factors
- Sudden deceleration, pivoting, or change in direction.
- Landing from a jump with knee in valgus/rotation.
- Direct blow to lateral knee causing valgus stress.
Risk Factors
- Female sex (smaller notch width, hormonal effects, neuromuscular control differences).
- High-risk sports: soccer, basketball, netball, skiing.
- Poor conditioning or hamstring–quadriceps imbalance.
- Previous ACL injury.
Pathophysiology
- High-energy force → valgus + external rotation or hyperextension.
- ACL fibres fail → partial or complete tear.
- Immediate hemarthrosis from ligament vascular supply.
- Knee instability → recurrent giving-way episodes.
- Long-term: meniscal tears, chondral damage, osteoarthritis.
ACL injury is both an acute stabiliser failure and a long-term risk factor for degenerative joint disease.
Clinical Manifestations
- “Pop” at injury, immediate swelling within 2–3 hours (hemarthrosis), inability to continue playing.
- Instability (“giving way”), difficulty pivoting.
Examination
- Positive Lachman test (most sensitive).
- Positive Anterior drawer test.
- Positive Pivot shift test (most specific).
- Reduced ROM due to swelling.
Rapid hemarthrosis + pop + instability strongly suggests ACL tear.
Unhappy Triad of O’Donoghue: ACL tear, MCL tear, Medial meniscus tear.
Diagnosis
- Clinical: History + exam (Lachman = gold standard).
- MRI: Imaging of choice; sensitivity >95%, shows associated meniscal or chondral injuries.
- X-ray: Rule out fractures (Segond fracture = pathognomonic for ACL tear).
Differential Diagnosis
| Condition | Differentiating Feature |
| Meniscal tear | Locking/catching, delayed swelling, positive McMurray |
| MCL tear | Medial pain, valgus stress laxity |
| PCL tear | Posterior sag sign, mechanism = dashboard injury |
Treatment
- Acute management: RICE (rest, ice, compression, elevation), analgesia, knee brace, physiotherapy.
- Non-operative: Activity modification + rehab (quads/hamstring strengthening). Suitable for low-demand or older patients.
- Operative: Arthroscopic ACL reconstruction (hamstring/patellar tendon autograft). Indicated in young, active patients, recurrent instability, or associated meniscal/chondral injury.
- Rehabilitation: Gradual return to sport after 9–12 months, with neuromuscular training.
Young + active = surgery; older + low-demand = physiotherapy.
Complications & Prognosis
- Recurrent instability if untreated.
- Secondary meniscal tears.
- Early osteoarthritis.
- Arthrofibrosis (post-surgical).
- Graft failure after reconstruction.
- Good with surgical reconstruction + rehab
- High return to sport but increased long-term OA risk.
Even with reconstruction, ACL injury predisposes to OA.
References
- Griffin LY, Albohm MJ, Arendt EA, et al. Understanding and preventing noncontact ACL injuries. Am J Sports Med. 2006;34(9):1512–32.
- van Eck CF, Schkrohowsky JG, Working ZM, et al. Clinical examination in the diagnosis of ACL tears: a meta-analysis. Knee Surg Sports Traumatol Arthrosc. 2013;21(9):1989–96.
- Frobell RB, Roos HP, Roos EM, et al. Treatment for acute ACL tear: 5-year outcome of the randomized controlled trial. N Engl J Med. 2010;363:331–42.
- Filbay SR, Grindem H. Evidence-based recommendations for rehabilitation after ACL reconstruction. Curr Rev Musculoskelet Med. 2019;12(2):289–96.
- Sanders TL, Maradit Kremers H, Bryan AJ, et al. Incidence of ACL tears and reconstruction: a 20-year population-based study. Am J Sports Med. 2016;44(6):1502–7.













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